๐Ÿ—‚ ็ธฝ็›ฎ้Œ„ ๏ฝœ ๐Ÿ“– ่‹ฑๆ–‡ๅŽŸๆ–‡๏ผˆๆœฌ็ฏ‡๏ผ‰ ๏ฝœ ๐Ÿ“ ๅฎŒๆ•ด็ฟป่ญฏ ๏ฝœ โญ ็ฒพ่ฏ็ญ†่จ˜

GINGIVAL HYPERPLASIA

GINGIVAL HYPERPLASIA

โ€ข Findings: Hypertrophy of both the free and attached gingivae, particularly the interdental papillae (Fig. 33-9).
โ€ข Inflammatory enlargement: Most common cause of gingival enlargement. Caused by edema and infective cellular infiltration caused by prolonged exposure to bacterial plaque; fibrosis occurs if untreated.
โ€ข Drug-induced fibrous hyperplasia of gingivae: May cover the teeth and is associated with:
โ€ข Anticonvulsants: Phenytoin, succinimides, and valproic acid.
โ€ข Calcium channel blockers: Nifedipine and verapamil.
โ€ข Cyclosporine.
โ€ข Systemic conditions/disorders:
โ€ข Pregnancy, puberty, vitamin C deficiency, and glycogen storage disease.
โ€ข Chronic myelomonocytic leukemia (Fig. 33-9).

FIGURE 33-9 โ€ข Gingival hyperplasia: Acute monocytic leukemiaโ€‚The gingivae show hyperplasia resulting from infiltration with leukemic monocytes.